Ethnic origin has been reported to affect the prevalence of atopic diseases in several studies in different parts of the world.
However, little is known about the prevalence of asthma and atopy in immigrants living in Europe.
The objective of this study was to evaluate the prevalence of asthma and atopy in Turkish children living in Germany and to investigate the role of ethnic origin on the development of asthma and atopy in this population.
In a cross-sectional survey the prevalence of physician-diagnosed asthma, atopy, skin-prick tests and bronchial hyperresponsiveness (BHR) to cold dry air challenge was assessed in 7,445 school children aged 9-11 yrs, living in Munich, south Germany.
Questionnaires were distributed to the parents for self-completion and children underwent skin prick tests and cold air hyperventilation challenge.
The Turkish children showed a significantly lower prevalence of asthma (5.3 versus 9.4%, p<0.05) than their German peers.
Furthermore, atopy, as assessed by skin prick tests (24.7 versus 36.7%, p<0.001) and BHR (3.9 versus 7.7%, p<0.001), was less common in Turkish children.
In multivariate regression models controlling for potential explanatory factors, Turkish origin still showed a significantly lower risk of developing asthma, atopic sensitization and BHR.
The prevalence of childhood asthma was therefore shown to be lower in Turkish children living in Germany than in Turkey. (...)
Mots-clés Pascal : Asthme, Enfant, Homme, Prévalence, Atopie, Turquie, Asie, Allemagne, Europe, Origine ethnique, Immigration, Exploration, Etude comparative, Appareil respiratoire pathologie, Bronchopneumopathie obstructive, Immunopathologie, Allergie
Mots-clés Pascal anglais : Asthma, Child, Human, Prevalence, Atopy, Turkey, Asia, Germany, Europe, Ethnic origin, Immigration, Exploration, Comparative study, Respiratory disease, Obstructive pulmonary disease, Immunopathology, Allergy
Notice produite par :
Inist-CNRS - Institut de l'Information Scientifique et Technique
Cote : 99-0280823
Code Inist : 002B11B. Création : 16/11/1999.