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  1. Acute symptomatic seizure disorders in young children : A population study in Southern Taiwan.

    Article - En anglais


    To determine the incidence, etiology, and prognosis of acute symptomatic seizures in children by age 3 years.


    In a population-based birth cohort study of all 15,209 neonatal survivors born in Tainan City between October 1989 and December 1991, parents or caretakers of 13,493 children aged 3 years were surveyed by telephone regarding any provoked convulsive disorder, particularly acute symptomatic seizure, in the children : medical records were reviewed.


    Sixty-three children (39 boys, 24 girls) had acute symptomatic seizures (incidence 0.46 in 100).

    The leading causes of acute symptomatic seizures were acute gastroenteritis, encephalitis/encephalopathy, and bacterial meningitis.

    Age-specific incidence was highest in the group aged 1-12 months.

    Intracranial hemorrhage. bacterial meningitis, and metabolic disturbance were the major causes of acute symptomatic seizures in children aged 1-12 months.

    Acute gastroenteritis, encephalitis/encephalopathy and bacterial meningitis accounted for 85% of the causes in children aged 13-24 months, and gastroenteritis and encephalitis/encephalopathy were the predominant causes in those aged 25-36 months.

    By age 5 years. subsequent unprovoked seizures developed in 14% of the survivors of acute symptomatic seizures.


    Many acute symptomatic seizures are preventable.

    The risk of subsequent unprovoked seizures is determined by underlying precipitating factors. (...)

    Mots-clés Pascal : Convulsion, Aigu, Taiwan, Asie, Sud, Incidence, Epidémiologie, Etiologie, Pronostic, Enfant, Homme, Trouble neurologique

    Mots-clés Pascal anglais : Convulsion, Acute, Taiwan, Asia, South, Incidence, Epidemiology, Etiology, Prognosis, Child, Human, Neurological disorder

    Logo du centre Notice produite par :
    Inist-CNRS - Institut de l'Information Scientifique et Technique

    Cote : 98-0430914

    Code Inist : 002B17A03. Création : 25/01/1999.