Temporal and spatial distributions of ozone in Atlanta : Regulatory and epidemiologic implications.
Relationships between ambient levels of selected air pollutants and pediatric asthma exacerbation in Atlanta were studied retrospectively.
As a part of this study, temporal and spatial distributions of ambient ozone concentrations in the 20-county Atlanta metropolitan area during the summers of 1993,1994, and 1995 were assessed.
A universal kriging procedure was used for spatial interpolation of aerometric monitoring station data.
In this paper, the temporal and spatial distributions of ozone are described, and regulatory and epidemiologic implications are discussed.
For the study period, the Atlanta ozone nonattainment area based on the 1-h, exceedance-based standard of 0.12 ppm is estimated to expand - from 56% of the Atlanta MSA by area and 71% by population to 88% by area and 96% by population - under the new 8-h, concentration-based standard of 0.08 ppm.
Regarding asthma exacerbation, a 4% increase in pediatric asthma rate per 20-ppb increase in ambient ozone concentration was observed (p-value=0.001), with ambient ozone level representing a general indicator of air quality due to its correlations with other pollutants.
The use of spatial ozone estimates in the epidemiologic analysis demonstrates the need for control of demographic covariates in spatiotemporal assessments of associations of ambient air pollutant concentrations with health outcome.
Mots-clés Pascal : Géorgie, Etats Unis, Amérique du Nord, Amérique, Atlanta, Zone urbaine, Pollution air, Qualité air, Inventaire source pollution, Ozone, Teneur air ambiant, Variation spatiale, Variation temporelle, Toxicité, Enfant, Homme, Asthme, Allergie, Epidémiologie, Appareil respiratoire pathologie, Bronchopneumopathie obstructive, Immunopathologie, O3
Mots-clés Pascal anglais : Georgia, United States, North America, America, Atlanta, Urban area, Air pollution, Air quality, Pollution source inventory, Ozone, Ambient air concentration, Spatial variation, Time variation, Toxicity, Child, Human, Asthma, Allergy, Epidemiology, Respiratory disease, Obstructive pulmonary disease, Immunopathology
Notice produite par :
Inist-CNRS - Institut de l'Information Scientifique et Technique
Cote : 98-0288968
Code Inist : 001D16C02. Création : 27/11/1998.