This paper reports findings from an ethnographic study of self-care behaviors and illness concepts among Mexican-American non-insulin dependent diabetes mellitus (NIDDM) patients.
Openended interviews were conducted with 49 NIDDM patients from two public hospital outpatient clinics in South Texas.
They are self-identified Mexican-Americans who have had NIDDM for at least I yr, and have no major impairment due to NIDDM.
Interviews focused on their concepts and experiences in managing their illness and their self-care behaviors.
Clinical assessment of their glucose control was also extracted from their medical records.
The texts of patient interviews were content analyzed through building and refining thematic matrixes focusing on their causal explanations and treatment behaviors.
We found patients'causal explanations of their illness often are driven by an effort to connect the illness in a direct and specific way to their personal history and their past experience with treatments.
While most cite biomedically accepted causes such as heredity and diet, they elaborate these concepts into personally relevant constructs by citing Provoking Factors, such as behaviors or events.
Their causal models are thus both specific to their personal history and consistent with their experiences with treatment success or failure.
Based on these findings, we raise a critique of the Locus of Control Model of treatment behavior prevalent in the diabetes education literature. (...)
Mots-clés Pascal : Diabète non insulinodépendant, Homme, Ethnie, Latinoaméricain, Comportement, Cause, Autoperception, Contrôle externe interne, Milieu culturel, Etats Unis, Amérique du Nord, Amérique, Endocrinopathie, Soin autogéré
Mots-clés Pascal anglais : Non insulin dependent diabetes, Human, Ethnic group, Latinamerican, Behavior, Cause, Self perception, Locus of control, Cultural environment, United States, North America, America, Endocrinopathy, Selfcare
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Inist-CNRS - Institut de l'Information Scientifique et Technique
Cote : 98-0195869
Code Inist : 002B30A03C. Création : 11/09/1998.