Percutaneous blood exposure among Danish doctors : Exposure mechanisms and strategies for prevention.
The objective of this study was to describe the mechanisms of percutaneous blood exposure (PCE) among doctors and discuss rational strategies for prevention.
Data were obtained as part of a nation-wide questionnaire survey of occupational blood exposure among hospital employed doctors in Denmark.
The doctors were asked to describe their most recent PCE, if any, within the previous 3 months.
Detailed information on the instruments, procedures, circumstances and mechanisms that caused the PCE was obtained.
Of 9375 doctors, 6256 (67%) responded, and 6005 questionnaires were eligible for analysis.
Of 971 described PCE the majority were caused by suture needles (n=483), IV-catheter-stylets (n=94), injection needles (n=75), phlebotomy needles (n=53), scalpels (n=45), arterial blood sample needles (n=41) and bone fragments (n=23).
Inattentiveness was the most common cause, contributing to 30.5% of all PCE.
Use of fingers rather than instruments was a major cause of injury in surgical specialties and was a contributing cause of 36.9% PCE on suture needles.
Common contributing causes when fingers were used (n=199) were poor space in (30.2%) or view of (18.6%) the operation field.
It was often argued that instruments were not practical to use or might harm the tissue.
Of 689 PCE in surgical specialties, 17.4% were inflicted by colleagues.
Up to 53. (...)
Mots-clés Pascal : Exposition professionnelle, Médecin, Sang, Voie percutanée, Epidémiologie, Incidence, Facteur risque, Prévention, Homme, Danemark, Europe, Médecine travail, Education santé, Personnel sanitaire, Questionnaire
Mots-clés Pascal anglais : Occupational exposure, Physician, Blood, Percutaneous route, Epidemiology, Incidence, Risk factor, Prevention, Human, Denmark, Europe, Occupational medicine, Health education, Health staff, Questionnaire
Notice produite par :
Inist-CNRS - Institut de l'Information Scientifique et Technique
Cote : 97-0432261
Code Inist : 002B30B01B. Création : 19/12/1997.