Al Eskan disease : Persian Gulf syndrome.
This article examines the potential relationship between Al Eskan disease and the Persian Gulf syndrome.
Al Eskan disease, reported in Military Medicine in 1992, is a novel and previously unreported condition triggered by the exceptionally fine sand dust of the Central and Eastern Saudi Arabian peninsula.
We repeat our study of the pathogenesis of Al Eskan disease to include the ultrastructural and microanalytical study of the sand, aerobiological studies of the kingdom of Saudi Arabia, and the etiology, symptoms, and prevalence of the disease.
We conclude that immunodepression resulting from the continued presence of sand particles less than 1mum in diameter in the lungs and bodies of Persian Gulf veterans explains not only the symptoms of the hyperergic lung condition of phase I and file symptoms of phase II of Al Eskan disease, but also provides an important clue to a common factor in most cases of Persian Gulf illnesses.
We include a discussion of most of the commonly suspected agents in the Persian Gulf syndrome.
In this case, we conclude that each of these factors, such as oil well fires, old-world diseases, or depleted uranium, are probably adjuvant or contributing causes. (...)
Mots-clés Pascal : Golfe Persique, Syndrome, Historique, Physiopathologie, Exploration microbiologique, Dépistage, Sable, Etiopathogénie, Forme clinique, Diagnostic différentiel, Prévention, Article synthèse, Homme, Arabie Saoudite, Asie, Pronostic, Ancien combattant, Immunopathologie, Infection, Appareil respiratoire pathologie, Al Eskan Disease.
Mots-clés Pascal anglais : Persian Gulf, Syndrome, Case history, Pathophysiology, Microbiological investigation, Medical screening, Sand, Etiopathogenesis, Clinical form, Differential diagnostic, Prevention, Review, Human, Saudi Arabia, Asia, Prognosis, Veteran, Immunopathology, Infection, Respiratory disease
Notice produite par :
Inist-CNRS - Institut de l'Information Scientifique et Technique
Cote : 97-0137077
Code Inist : 002B06C05. Création : 21/05/1997.