A consideration of the effects of residential mobility produces much more realistic estimates of typical individuals'radon exposures and mortality risks than those of the Environmental Protection Agency (EPA).
A model linking residential mobility, the distribution of radon in US homes, and lung cancer risk is used to simulate lifetime radon exposure, with and without mitigation of high-radon homes, for typical mobile individuals.
Radon-related lung cancer mortality risks are then estimated for smokers and never-smokers.
Most individuals residing in high-radon homes have equivalent lifelong radon exposures well below those they are currently experiencing.
Consequently, actual lung cancer risks are generally well belwo those implied in the EPA's radon risk charts.
For most people who mitigate high-randon homes, risk reduction is modest.
Radon may indeed be responsible for as large a population risk of lung cancer as the EPA estimates.
However, caution must be used in interpreting the EPA's risk assessment for individuals ; in many cases, mitigation will have little effect on residents'health risks.
Mots-clés Pascal : Tumeur maligne, Bronchopulmonaire, Radon, Effet biologique, Logement habitation, Exposition, Facteur risque, Toxicité, Mortalité, Homme, Santé et environnement, Epidémiologie, Etats Unis, Amérique du Nord, Amérique, Appareil respiratoire pathologie, Poumon pathologie, Bronche pathologie
Mots-clés Pascal anglais : Malignant tumor, Bronchopulmonary, Radon, Biological effect, Housing, Exposure, Risk factor, Toxicity, Mortality, Human, Health and environment, Epidemiology, United States, North America, America, Respiratory disease, Lung disease, Bronchus disease
Notice produite par :
Inist-CNRS - Institut de l'Information Scientifique et Technique
Cote : 96-0452603
Code Inist : 002B11A. Création : 10/04/1997.