The first methylmercury poisoning by consumption of fish arose in Minamata, Japan, in 1953.
Methylmercury dispersed from Minamata to the Shiranui Sea until 1968.
Mercury concentration in the hair of residents on the coast of the Shiranui Sea was 10 to 20 times higher than that in nonpolluted people in Kumamoto Prefecture in 1960.
People on the coast of the Shiranui Sea have consumed fish containing low-dose methylmercury without a ban over decades until 1968.
We studied the effect of long-term consumption of methylmercury on those people 10 years later after the end of methylmercury dispersion.
Our epidemiological study clarified that people in a fishing village (Ooura) on the coast of the Shiranui Sea showed a significantly higher frequency of neurological signs characteristic of methylmercury poisoning (hypoesthesia, ataxia, impairment of hearing, visual change, and dysarthria) in comparison with people in a nonpolluted fishing village (Ichiburi).
The neurological disorders were still detected 10 years later in Ooura after the end of methylmercury dispersion from Minamata ; hypoesthesia showed the highest frequency in Ooura.
These results suggest that people on the coast of the Shiranui Sea were affected by long-term dietary exposure to methylmercury.
Mots-clés Pascal : Pollution eau, Métal lourd, Contamination, Poisson comestible, Toxicité, Homme, Système nerveux pathologie, Epidémiologie, Long terme, Zone côtière, Japon, Asie, Pêche, Milieu rural, Mercure(méthyl)
Mots-clés Pascal anglais : Water pollution, Heavy metal, Contamination, Edible fish, Toxicity, Human, Nervous system diseases, Epidemiology, Long term, Coastal zone, Japan, Asia, Fishing, Rural environment
Notice produite par :
Inist-CNRS - Institut de l'Information Scientifique et Technique
Cote : 96-0240217
Code Inist : 002B03M03. Création : 199608.